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Exercise-induced syncope and Brugada syndrome


1 Department of Pediatric Cardiology, University of California-Irvine and Children's Hospital of Orange County, Orange CA, USA
2 Department of Pediatrics, University of California-Irvine and Children's Hospital of Orange County, Orange CA, USA

Correspondence Address:
Dr. Anjan S Batra
1140 W. La Veta Ave. Suite 750, Orange, CA 92868
USA
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Source of Support: None, Conflict of Interest: None


DOI: 10.4103/apc.APC_131_18

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Year : 2019  |  Volume : 12  |  Issue : 3  |  Page : 292-294

 

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Brugada syndrome (BrS) is a hereditary condition that is characterized by ST elevation, ventricular tachycardia or fibrillation, and sudden cardiac death in otherwise healthy patients. Life-threatening arrhythmias generally occur, while at rest, with fever or during vagotonic states. Exercise is generally not considered a trigger for ventricular arrhythmias or syncope in patients with BrS. We describe a patient who presented with exercise-induced syncope, ventricular tachycardia during an exercise test, and was found to be both genotypically and phenotypically positive for BrS. This case highlights a potentially important role of exercise testing in diagnosing and risk stratifying certain patients with BrS.






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1 Department of Pediatric Cardiology, University of California-Irvine and Children's Hospital of Orange County, Orange CA, USA
2 Department of Pediatrics, University of California-Irvine and Children's Hospital of Orange County, Orange CA, USA

Correspondence Address:
Dr. Anjan S Batra
1140 W. La Veta Ave. Suite 750, Orange, CA 92868
USA
Login to access the Email id

Source of Support: None, Conflict of Interest: None


DOI: 10.4103/apc.APC_131_18

Rights and Permissions

Brugada syndrome (BrS) is a hereditary condition that is characterized by ST elevation, ventricular tachycardia or fibrillation, and sudden cardiac death in otherwise healthy patients. Life-threatening arrhythmias generally occur, while at rest, with fever or during vagotonic states. Exercise is generally not considered a trigger for ventricular arrhythmias or syncope in patients with BrS. We describe a patient who presented with exercise-induced syncope, ventricular tachycardia during an exercise test, and was found to be both genotypically and phenotypically positive for BrS. This case highlights a potentially important role of exercise testing in diagnosing and risk stratifying certain patients with BrS.






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