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Figure 1: (a) Active phase of TA in descending thoracic aorta showing a granulomatous inflammation at the medio-adventitial junction of aorta destroying the structure of an intercostal artery; (b) chronic phase of TA in descending thoracic aorta showing media (M) at the outer third with clusters of mononuclear cells. Note marked fibrosis of the adventitia (A) (Hematoxylin and Eosin, ×250). (c) The cells are lymphocytes, and few plasma cells and histiocytes (Hematoxylin and Eosin, ×250)

Figure 1: (a) Active phase of TA in descending thoracic aorta showing a granulomatous inflammation at the medio-adventitial junction of aorta destroying the structure of an intercostal artery; (b) chronic phase of TA in descending thoracic aorta showing media (M) at the outer third with clusters of mononuclear cells. Note marked fibrosis of the adventitia (A) (Hematoxylin and Eosin, ×250). (c) The cells are lymphocytes, and few plasma cells and histiocytes (Hematoxylin and Eosin, ×250)